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Shuren, X. (1994). An introduction to feeding and management of snow leopard in Xining Zoo, China. In J.L.Fox, & D.Jizeng (Eds.), (pp. 177–182). Usa: Islt.
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Doherty, J., & Wharton, D. (1988). Breeding Management of the Snow Leopard at the New York Zoological Park. In H.Freeman (Ed.), (pp. 173–179). India: International Snow Leopard Trust and Wildlife Institute of India.
Abstract: Since 1996, the Bronx Zoo has produced 48 snow leopard cubs. Some of the cubs born in the last several years are the great-great grandchildren of the Bronx male Sherpa born in 1966. These five generations have all been bred and reared in the Zoological Park's Lion House which opened in 1903......Instead we will focus primarily on mangement and manipulation for cub production.
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Isenbugel, E., & Weilemann P. (1988). Treatment of Bladder Diverticulum and Ascites in a Female Snow Leopard. In H.Freeman (Ed.), (pp. 171–172). India: International Snow Leopard Trust and Wildlife Institute of India.
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Gurung, C. P. (1997). Ecotourism: Nepal's Experience. In R.Jackson, & A.Ahmad (Eds.), (pp. 170–177). Lahore, Pakistan: Islt.
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Pollock, R. V., & Carmichael, L. E. (1983). Use of modified live feline panleukopenia virus vaccine to immunize dogs against canine parvovirus. Am J Vet Res, 44(2), 169–175.
Abstract: Modified live feline panleukopenia virus (FPLV) vaccine protected dogs against canine parvovirus (CPV) infection. However, unlike the long- lived (greater than or equal to 20-month) immunity engendered by CPV infection, the response of dogs to living FPLV was variable. Doses of FPLV (snow leopard strain) in excess of 10(5.7) TCID50 were necessary for uniform immunization; smaller inocula resulted in decreased success rates. The duration of immunity, as measured by the persistence of hemagglutination-inhibiting antibody, was related to the magnitude of the initial response to vaccination; dogs with vigorous initial responses resisted oronasal CPV challenge exposure 6 months after vaccination, and hemagglutination-inhibiting antibodies persisted in such dogs for greater than 1 year. Limited replication of FPLV in dogs was demonstrated, but unlike CPV, the feline virus did not spread to contact dogs or cats. Adverse reactions were not associated with living FPLV vaccination, and FPLV did not interfere with simultaneous response to attenuated canine distemper virus.
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Trepanier, L. A., Cribb, A. E., Spielberg, S. P., & Ray, K. (1998). Deficiency of cytosolic arylamine N-acetylation in the domestic cat and wild felids caused by the presence of a single NAT1-like gene. Pharmacogenetics, 8(2), 169–179.
Abstract: The purpose of this study was to determine the molecular basis for a relative deficiency in the cat of cytosolic arylamine N- acetyltransferase (NAT), an enzyme family that is important in the metabolism of xenobiotics and that normally consists of at least two related enzymes, NAT1 and NAT2. N-acetyltransferase in feline liver showed high affinity (mean Km = 2.1 microM) for p-aminobenzoic acid, an NAT1 selective substrate in humans and rabbits, but showed a very poor affinity (mean Km > 10 mM) for sulfamethazine, an NAT2 selective substrate in humans and rabbits. Immunoreactive N-acetyltransferase was detected in feline liver, bladder and colon using an NAT1-specific antipeptide antibody, but was not detected in any tissues using an NAT2- specific antibody. Southern blot analysis of genomic DNA demonstrated a single band in domestic cats using each of six restriction digests; single bands were also found on Southern blot analysis of six wild felids. The deduced amino acid sequence of the central portion of feline N-acetyltransferase, obtained by polymerase chain reaction amplification in both domestic cats and seven wild felids (lion, tiger, lynx, snow leopard, bobcat, Asian leopard cat and cheetah), contained three residues, Phe125, Arg127, and Tyr129, which determine NAT1-like substrate specificity in humans. These results support the conclusion that cytosolic arylamine N-acetylation activity is low in the cat because of the presence of a single N-acetyltransferase that has substrate specificity, immunogenicity and sequence characteristics similar to human NAT1, and that the unusual presence of only a single N- acetyltransferase gene appears to be a family wide trait shared by other felids.
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Fox, J. L., Nurbu, C., & Chundawat, R. S. (1991). The Mountain Ungulates of Ladakh India. Biological Conservation, 58, 167–190.
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Yanfa, L. (1994). The care, breeding and diseases of snow leopards in Qinghai, China. In J.L.Fox, & D.Jizeng (Eds.), (pp. 167–175). Usa: Islt.
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Schmidt, R. E., Eisenbrandt, D. L., & Hubbard, G. B. (1984). Tyzzer's disease in snow leopards. J Comp Pathol, 94(1), 165–167.
Abstract: Tyzzer's disease was diagnosed histologically in 2 litters of newborn snow leopard kittens. The gross and histological lesions were similar to those reported in domestic cats and other animals. No signs of illness was noted in either of the snow leopard dams.
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Jalanka, H. H. (1989). Evaluation and comparison of 2 ketamine-based immobilization techniques in snow leopards (Panthera uncia). Journal of Zoo and Wildlife Medicine, 20(2), 163–169.
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