Wack, R. F., & Kramer, L. W. (1995). Multifocal osteomyelitis in a young snow leopard (Panthera uncia). Journal of Zoo and Wildlife Medicine, 26(4), 553–563.
Abstract: A 5-mo-old male snow leopard (Panthera uncia) was presented for mild rear leg lameness. Osteomyelitis was suspected on the basis of radiographic changes and confirmed by histopathology of a biopsy sample from the affected bone. Aerobic cultures of the biopsies repeatedly grew Klebsiella oxytoca. Repeated anaerobic and fungal cultures did not result in growth. The leopard was treated unsuccessfully with cefadroxil, chloramphenicol, and trimethoprim/sulfadiazine despite apparent in vitro sensitivity to these antibiotics. Successful resolution was eventually achieved with enrofloxacin, 7.5 mg/kg p.o. b.i.d. for 60 days. The number of bones involved (right humerus, right and left ulna, right and left radius, right and left femur, right and left tibia, mandible, right metatarsus) made this an unusual presentation of osteomyelitis.
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Wahlberg, C., Tarkkanen, A., & Blomqvist, L. (1982). Further observations on the multiple ocular coloboma (MOC) in the snow leopard, Panthers uncia. In L. Blomqvist (Ed.), International Pedigree Book of Snow Leopards (Vol. 3, pp. 139–144). Helsinki: Helsinki Zoo.
Abstract: The first observation of the occurrence of multiple ocular coloboma (MOC) in a snow leopard was reported in the International Pedigree Book of Snow Leopards Volume I in 1978 (1). The lesions in this syndrome consist of coloboma of the upper eye lid and uveal coloboma of the globe. Even colobomatous retinal cysts and retinal dysplasia have been noted. The ethiology of in all ten cases of MOC in the snow leopards kept at the Helsinki Zoo were described and discussed in detail in Volume II of the International Pedigree Book of Snow Leopards (2,3). Three cases of MOC in the snow leopards kept at Henry Doorly Zoo, Omaha, Ne., have been described by Phillips (4), one case is known of in Amsterdam (van Bree, personal communication), and two cases in Zoo Zurich (Isenbugel and Weilenmann, pers. comm.) The ethiology of the defect is still not known although various theories ranging from genetic to exogenous factors have been presented.
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